More than 50% of women experience hairloss at some stage in their lifetime...

Hair loss in Women

Boost n Blend hair building fibres cover visible scalp for women with hair loss

You are not alone

More than 50% of women will experience hair loss at some point in their lifetime1. Yes, you read that correctly, more than HALF! This makes female hair loss just as common as having sensitive skin2 or wearing glasses3 so you’re certainly not alone.

So why do millions of Australian women suffer from hair loss? We've outlined a number of potential causes, but if your hair loss has got you worried, please consult a doctor. 

For a full description of Pregnancy

and Hair loss click here.

Hereditary Hair Loss

Hereditary hair loss, also known as androgenetic alopecia, is the most common cause of hair loss4 in women. Hair growth is cyclical, passing through three main stages: the growth stage (anagen phase), the rest stage (catagen phase) and then finally, the shedding stage (telogen phase).

Each individual hair may endure on the scalp for three to seven years before entering the catagen phase for around two weeks and then the telogen phase which can last for a few months. It is during this final stage that hair begins to sit higher on the scalp and can be pulled out with very little effort such as when brushing or washing your hair. After the hair has been shed, a new one will take its place and begin to grow. In the case of hereditary hair loss, this cycle becomes a lot quicker and it may only take months for a new hair to be shed, or in some cases a new hair may not replace the recently shed hair at all.

Like other hereditary conditions, hereditary hair loss is caused not only by genetic predisposition, but by your genes in conjunction with your hormones. This leads us to telogen effluvium, another common cause of hair loss in women.

Telogen Effluvium

Like hereditary hair loss, telogen effluvium is extremely common. This phenomenon occurs when your body experiences some form of change such as pregnancy, major surgery, drastic weight loss or even extreme stress can all cause you to experience hair loss. After bone marrow, hair is the second fastest growing tissue of the body5, therefore any small changes in your body are likely to be reflected in your hair.

Boost n Blend hides visible scalp for hair loss in women

Now you may be thinking, “but I’m not experiencing any of these changes!”. Well keep in mind that due to the various phases of the hair cycle, it may take weeks or even months for the hair to be affected, so if you believe this may be the cause of your hair loss, look back a few months and you may just find the culprit.

In most cases where hair loss is a result of telogen effluvium it’s a case of simply waiting for your body to regain equilibrium. The good news is that whilst you wait, BOOSTnBLEND® can ensure that no one notices a thing!  

Low Iron/B12

As with telogen effluvium, changes in vitamins and minerals in your body can lead to hair loss and this is especially apparent when it comes to iron stores and B12. A quick trip to the doctor for some blood tests for B12 and serum feritin (iron stores) will reveal whether or not this could be contributing to your hair loss. The good news about this particular cause for hair loss is that like telogen effluvium, when the appropriate iron and B12 levels are restored through the use of supplements and/or dietary changes, your hair should return to normal. In the meantime, however, using BOOSTNBLENDTM to cover any areas of visible scalp will ensure your issue remains private!



[1] Quan Dinh and Rodney Sinclair, 'Female pattern hair loss: Current treatment concepts. ' (2007) 2 Clinical Interventions in Aging 189

[2] J Escalas-Taberner, E González-Guerra and A Guerra-Tapia, 'Sensitive Skin: A Complex Syndrome' (2011) 102(8) Actas Dermo-Sifiliográficas (English Edition) 563.

[3] http://www.fahcsia.gov.au/our-responsibilities/women/publications-articles/general/women-in-australia/women-in-australia-2007?HTML#population

[4] Jerry Shapiro, 'Hair Loss in Women' (2007) 357(16) New England Journal of Medicine 1620.

[5] Ibid.